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Metabolism of type III collagen, the major constituent of many dense and most loose connective tissues, except bone, tendon and cartilage, can be measured by serum aminoterminal propeptide of type III procollagen (PIIINP). PIIINP reßects the overall metabolism of all soft connective tissues in the body (Risteli et al. Serum levels of ICTP and PIIINP are reported to increase during hormone replacement therapy with nandrolone decanoate (Hassager et al. This is thought to be due to increased breakdown of collagen types I and III in non-bone tissues. ICTP was suggested to derive relatively more from nonbone tissues than from bone because it did not correlate with histomorphometric measurements of bone turnover (Hassager et al. AAS at high concentrations may also bind to glucocorticoid receptors, and thus, AAS may have an impact on collagens by counteracting the effect of corticosteroid (Rogol & Yesalis 1992, Haupt 1993). AAS may also inßuence collagen metabolism by direct stimulation of androgen receptors, which are known to be present in low densities in osteoblasts and bone marrow, thus also inderectly inhibiting osteoclast precursors and bone resorption.

 

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